I agree BW test is fine for a first cycle AND if you keep the dose at or below 500mg/week (which is actually only TWICE that used for TRT) PCT is also unlikely to be needed especially considering your age of TWENTY THREE. So again providing you cycle TT at a dose less than 500mg/week your "NO PCT" HTPA recovery, God forbid I even suggest such a thing, will be uneventful!
Finally although you would need to pin at least twice a week vs once a week if T-prop is chosen rather than T-c or T-e, HTPA recovery requires only half the time if T-p is selected because of their differing half lives (2-3 VS 6-8 DAYS)
I also agree although an Ai is unlikely to be needed, (but should available) IF your TT dose remains less than 500mg per week.
Finally since there is NO EVIDENCE progesterogenic AAS (Bolo, Tren or Deca) raise serum prolactin or progesterone levels (UNLESS your breast feeding pregnant or have a prolactin secreting tumor) the use of dopamine agonists such as Gabergoline or Bromocriptine is unwarranted.
Incidentally, I appreciate your questions but it's blatantly CLEAR you have A LOT more reading to do fella, in part since there is so much to know in this field of AAS yet also because there are MANY WAYS to run a cycle, prevent side AAS effects or use PCT and in part because your questions are rather fundamental but typical for someone whose cycling experience is limited.
Why is that? Because the evidence is sparse in certain areas and is often extrapolated from other indirect scientific studies.
For instance where does ALL the supportive PCT literature come from? I mean did some contemporary physician prescribe AAS to his BB patients and thereafter develop different means of weaning them off THUS restoring their HTPA? Was this data eventually published? Or how about these AI's people use for Estrogen side effects?
Of course the answer to the first question is NO since the prescribing of AAS as PED's is ILLEGAL and can cost a physician his license! So what then?
Well a considerable portion of existing PCT recommendations are extrapolated from males attempting to recover from "low T" for various reasons such as prostate cancer therapy, testicular failure, testicular tumors, infertility etc! So these patients actually USED "PCTherapy" as a means to recover the gonads or HTPA.
Now obviously some of the contrasts and comparisons may be INAPPROPRIATELY extrapolations and therefore NOT applicable to BB using PEDs.
For instance what is the data that supports the use of TWO SERMS? What studies was that therapy based on? Are their any one vs two SERM comparisons? (Nope) Needless to say I think the evidence approaches ZIPPO and some would disagree with my assertion.
However, I have my reasons and they have theirs, BUT those conflicting views should be EVIDENCE BASED, and that is the reason YOU need to read more. To develop YOUR OWN opinion based on the literature rather than parroting what I or someone else suggests as "the truth"!
Best
JIM
Oh most of the evidence on AI's AND SERMS is derived from females with BREAST CANCER! And we are using those studies for male BB on mega doses of AAS?
Yep! Why? Because it's the best we have, and one hell of an improvement over RATS,