Very nice study Hawk, although I'm not sure this applies to humans. I have a very long study in the form of a powerpoint presentation that I'm not sure how to put on here. It basically discusses that the human body has a finite number of receptors for each hormone and each hormone has a different binding affinity for that receptor. The amount of receptors has not been shown to increase with site specific AAS use.
However, the use of IGF-1 LR3 site specifically has been shown to induce cell duplication and mutation (hyperplasia) at larger doses. Increasing the number of cells would increase the number of receptor sites, logically speaking. Straight AAS use induces muscle cell growth, but NOT duplication. So, it would seem likely that gains made with peptides, slin, gh, etc are far more permanent than AAS gains. With a lack of receptors available for each drug to occupy, you have to plan your cycles wisely. It would be great if there were an accurate way for us to know when the receptors were saturated dosage-wise for each drug. For example, Trenbolone has a higher binding affinity to the AR than Testosterone. So, when they compete for the same receptor, Tren wins more than test. But, Tren also attempts to bind to the PR, which leaves some room for the test to slide in. So, my thought being if someone got good gains from 1 gram of test per week, they could logically lower that test dose a couple hundred mg and add 400mg Tren to the cycle and come up with slightly to significantly better gains. Does that make sense?
If there were people interested in studying this kinda stuff that I think about on a daily basis, the answer to my question would most likely already be answered. Hmmm, I may have to pioneer something myself.
BTW, Hawk I meant no disrespect to your post and I was just trying to shed some light on the situation. From that info I would have to assume that hypertrophy caused by oil pressure within the muscle fibers is a cause of muscle cell expansion, not duplication. In which case the neumber of receptors would reamin the same. But, may be it works differently in humans. There's a reason the bodybuilders of today are so much bigger than the pros in years past.... I think Hyperplasia is the answer. All made possible by good ol slin, IGF, GH, peptides, etc.