Thanks for trying to find out I have a history of them and the doc has me on low dose estrogen for it so I was just curious.Honestly I don't think its been studied enough asked two O.B.s here at the hospital and they had no idea. I'd be more worried about HGH than some low androgenic drugs like Var or Deca
A primary factor which determines the "responsiveness" of COD to hormonal therapy is whether they are "functional cysts".
However since the MOST COMMON ORIGIN of COD (in otherwise healthy females) is the development of FUNCTIONAL CYSTS from CYCLICAL ANOVULATION, AAS are more likely to LOWER their frequency, IMO!
On a comparative basis, BCP also cause annovulation, yet thru a different mechanism, by maintaining NON-CYCLICAL ELEVATION of E-2.
IMO the use of AAS would be MORE LIKELY to have the same benefit as BCP, since the hormonal environment would be similar, NON-CYCLICAL steady state, relatively low levels of estrogen.
Moreover this benefit has BEEN OBSERVED and STUDIED in patients using Danocrine for ENDOMETRIOSIS and COD!
It's primary MOA is to prevent the release of gonadotropins (LH and FSH), yet Danazol also has "mild" androgenic" effects.
Now whats important, Danocrine HAS been shown to decrease the frequency and reversed the presence of existing COD in some females.
regs
JIM
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