Research chems... Made my "rats" sick... ;-p
ALL of the side effects you mentioned have been reported with Gaber use, although their frequency and severity are directly proportional to the dosage.
On average the most common side effects are
GI : including vomiting, constipation and abdominal cramps
Neuro; Headache is noted in as many as 30%, visual alterations and hiccups
CV: Dizziness, palpitations
Since there are few conditions such as Parkinson's, or Pituitary adenoma which warrants the use of dopamine agonists, I query WHY some BB choose to use a drug which alters CNS neurotransmission.
Oh yea I've heard how these DA's combat the altered libido from a variety of PEDs.
However the fact is ONLY ONE CLINICAL STUDY has been performed to date in humans. The study was poorly controlled and limited in size to warrant the conclusion as cited by the authors "Gaber is effective in patients with anorgasmioa"!
Unfortunately many "bro scientists" Parrot what they have heard or read elsewhere, often citing this very study to support their opinion and extrapolate further by lamenting Gaber is effective for "many forms of sexual dysfunction"
Another reason some BB have elected to use Gaber is to lower "an elevated Prolactin level because of progesterogenic AAS". The genesis of this nonsense is/was apparently derived by combining research from several studies on pregnant patients, whom develop elevated prolactin and progesterone as a means to an ends, lactation.
References have also included patients with Prolactinomas, believing the mechanism responsible was as simplistic as "enhanced Progesterone receptor" stimulation rather than a deranged CNS milieu.
Others vainly cling to the well established data, that because the MOA of 19nor AAS is mediated via the progesterone receptor, they MUST also raise serum Progesterone / Prolactin levels, NOT!
There simply is no data which supports this broism, in spite of data (what little there is) to the contrary, which suggests 19nor AAS are more likely to LOWER Progesterone levels!
The bottom line, there is no evidence which supports the "labeled" or "off labeled" use of dopamine agonists (Bromocryptine, Gabergoline) as PED's or as a means of combating the complications of PEDs.
Regs
jim
As treatment for 19-Nor AAS complications? Heck no! If you suffer from gynecomastia while using these AAS the problem is invariably E-2 mediated and best treated with a SEEM!
Regs
Jim
What's up buddy! SERM for your typo.
Also, why the hell would you want to run a drug that supresses IGF-1/HGH secretion.
I would like to add most all situations can be managed with an AI. As the Doc suggest a SERM should be used if it gets to that nasty point.
mands
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